ACP-105 Vs AC-262536
Both ACP-105 and AC-262536 are partial agonists at the androgen receptor, however their anabolic:androgenic ratings are slightly different, with ACP-105 being more favorable with 67% anabolic activity : 21% androgenic activity as opposed to AC-262536 which exerted up to 66% of the anabolic benefits of Testosterone, while simultaneously only exerting 27% as much Androgenic activity.
Based on the statistics and interaction with the androgen receptor there isn’t a significant difference between the two, but ACP-105 seems to be a better choice when putting them head to head.
However, we do not know the binding affinity of ACP-105, while we know that the binding affinity of AC-262536 is Ki of 5nM.
I would still venture to guess that ACP-105 is a better choice for research that is geared towards maximizing anabolic activity with a minimization of potential side effects, however this could only be determined for certain with more data, or more anecdotal comparisons from credible sources.
Detection In Drug Tested Athletes
One thing that should be noted is that supposedly AC-262536 is still undetectable (so I’ve heard), so any drug-tested athlete would hypothetically be able to get a sizeable edge from AC-262536.
However, ACP-105 already is detectable and there has been a doping test designed to detect it.
I’d say if the research subject wasn’t engaging in tested sports, then ACP-105 would be the preferable choice in terms of sheer anabolic effect relative to potential suppression and side effects (based on the limited data available).
However, if the choice of the SARM used in research was in any way dictated by whether it could be detected or not, then AC-262536 would be the better choice in that particular scenario.
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